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Activation of phosphatidylinositol 3-kinase/AKT/snail signaling pathway contributes to epithelial-mesenchymal transition-induced multi-drug resistance to sorafenib in hepatocellular carcinoma cells.

机译：磷脂酰肌醇3-激酶/ aKT / snail信号传导途径的激活有助于上皮 - 间质转化诱导的肝细胞癌细胞中对索拉非尼的多药抗性。

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Jiejie Dong; Bo Zhai; Weihua Sun; Fengli Hu; Hao Cheng; Jun Xu;
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1. Activation of phosphatidylinositol 3-kinase/Akt signaling pathway mediates acquired resistance to sorafenib in hepatocellular carcinoma cells. [J] . Chen KF, Chen HL, Tai WT, The Journal of Pharmacology and Experimental Therapeutics: Official Publication of the American Society for Pharmacology and Experimental Therapeutics . 2011,第1期

机译：磷脂酰肌醇3-激酶/ Akt信号通路的激活介导肝细胞癌细胞对索拉非尼的获得性耐药。
2. Activation of phosphatidylinositol 3-kinase/Akt signaling mediates sorafenib-induced invasion and metastasis in hepatocellular carcinoma [J] . Wang Haiyong, Xu Litao, Zhu Xiaoyan, Oncology reports . 2014,第4期

机译：磷脂酰肌醇3-激酶/ Akt信号的激活介导索拉非尼诱导的肝癌侵袭和转移
3. LncRNA SNHG1 contributes to sorafenib resistance by activating the Akt pathway and is positively regulated by miR-21 in hepatocellular carcinoma cells [J] . Weidong Li, Xuesong Dong, Changjun He, Journal of experimental & clinical cancer research : . 2019,第1期

机译：LncRNA SNHG1通过激活Akt途径促进索拉非尼耐药，并在肝癌细胞中受到miR-21的正调控
4. Illuminating the Phosphatidylinositol 3-kinase/Akt Pathway [C] . Qiang Ni, Matthew Fosbrink, Jin Zhang Conference on Small Animal Whole-Body Optical Imaging Based on Genetically Engineered Probes; 20080121-22; San Jose,CA(US) . 2008

机译：阐明磷脂酰肌醇3-激酶/ Akt途径
5. Phosphatidylinositol 3-kinase/Akt signaling pathway and angiogenesis. [D] . Cao, Zongxian. 2006

机译：磷脂酰肌醇3-激酶/ Akt信号通路与血管生成。
6. Up-Regulation of Phosphatase in Regenerating Liver-3 (PRL-3) Contributes to Malignant Progression of Hepatocellular Carcinoma by Activating Phosphatase and Tensin Homolog Deleted on Chromosome Ten (PTEN)/Phosphoinositide 3-Kinase (PI3K)/AKT Signaling Pathway [O] . Bing-Hui Li, Yang Wang, Chao-Yang Wang, 1923

机译：再生肝脏3（PRL-3）中磷酸酶的上调通过激活被删除的磷酸酶和第十个染色体上的张力蛋白同源物（PTEN）/磷酸肌醇3-激酶（PI3K）/ AKT信号通路来促进肝细胞癌的恶性进展。
7. Up-Regulation of Phosphatase in Regenerating Liver-3 (PRL-3) Contributes to Malignant Progression of Hepatocellular Carcinoma by Activating Phosphatase and Tensin Homolog Deleted on Chromosome Ten (PTEN)/Phosphoinositide 3-Kinase (PI3K)/AKT Signaling Pathway [O] . Bing-Hui Li, Yang Wang, Chao-Yang Wang, 2018

机译：再生肝-3（PRL-3）中磷酶的上调通过在染色体10（PTEN）/磷酸阳性3-激酶（PI3K）/ Akt信号通路上激活磷酸酶和Tensin Homolog来有助于肝细胞癌恶性进展。

Activation of phosphatidylinositol 3-kinase/AKT/snail signaling pathway contributes to epithelial-mesenchymal transition-induced multi-drug resistance to sorafenib in hepatocellular carcinoma cells.

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